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Reperfusion Injury Market Report Explored in Latest Research 2024
by Metcarl · October 4, 2018
Reperfusion Injury Market: Description
Reperfusion injury is referred to as the tissue damage caused due to absence of blood supply or lack of oxygen such as damage to brain after stroke or heart post episodes of ischemia. Such kind of restoration of blood supply results into inflammation and oxidative damage through the induction of oxidative stress rather than normal functioning. Confined infarct size and reduced mortality can be achieved by prompt restoration of blood supply to myocardium. However, this restoration is also known to result in the cardiac damage and eventually reperfusion injury. A delay in reperfusion therapy has been attributed for such damage.
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Reperfusion of ischemic tissue is often associated with microvascular injury. The duration for which a tissue can survive deprived of oxygen varies, but eventually the tissue is rendered necrotic. The blood supply restoration usually minimizes the damage Basket Nike Air Vapormax Pas Cher , but reperfusion of tissue post thrombolytic procedures demonstrated that the extent of injury often increased on restoration of the blood supply. The studies have also attributed this reperfusion injury, to the activity of free radicals.
Reperfusion Injury Market: Segmentation
The global reperfusion injury market can be segmented based on therapy type, injury type and geography. Drugs used for treating reperfusion injury are classified on the basis of mode of action, target site Nike Air Vapormax Rose Cher , route of administration and molecule type. Geographically, the global reperfusion injury market can be categorized into four major regions namely, North America, Europe Nike Air Vapormax Bleu Pas Cher , Asia-Pacific and Rest of the World. Primary percutaneous coronary intervention (PCI) and thrombolysis, form the primary reperfusion therapies and are often used for treating acute coronary syndromes.
Reperfusion Injury Market: Overview
Allopurinol, one of the drugs in clinical trials helps inhibit production of free radicals thereby known to reduce the size of infarct. However the drug has not been universally successful. An enzyme called recombinant superoxide dismutase has been under investigation as free radical scavenger that detoxifies oxygen. Various other drug molecules in clinical trials include cyclosporine, MTP-131 Nike Air Vapormax Grise Pas Cher , bucillamine, recombinant proteins, type 5 adenylyl cyclase inhibitors, and synthetic peptides amongst others. Agents that specifically target proteins Nike Air Vapormax Blanche Pas Cher , transcription factors or ion channels, including PPAR, PKC agonistsantagonists, Phosphodiesterase-5 inhibitors Nike Air Vapormax Noir Pas Cher , the ATP-dependent potassium channel and 3-Hydroxy-3-methyl glutaryl coenzyme A reductase are also promising.
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